Paraneoplastic syndromes secondary to neuroendocrine tumours
نویسندگان
چکیده
منابع مشابه
Paraneoplastic syndromes secondary to neuroendocrine tumours.
Neuroendocrine tumours may be either benign or malignant tumours, and have the ability to synthesise and secrete biologically active substances characteristic of the cell of origin that can cause distinct clinical syndromes. The term 'paraneoplastic syndromes' (PNSs) is used to denote syndromes secondary to substances secreted from tumours not related to their specific organ or tissue of origin...
متن کامل[Associated gastroenteropancreatic neuroendocrine tumours to familiar syndromes].
Aproximately 5-10% of neuroendocrine tumours (NETs) of the gastroenteropancreatic system (GEP) have an hereditary background. The known hereditary syndromes include: multiple endocrine neoplasia type 1 (MEN 1), von Hippel Lindau disease (VHL), neurofibromatosis type 1 (NF 1) and tuberous sclerosis complex (TSC). This review discusses for each of these syndromes the: genes involved and specifics...
متن کاملAcute pancreatitis secondary to pancreatic neuroendocrine tumours.
CONTEXT Pancreatic neoplasms are an uncommon aetiology of acute pancreatitis. Pancreatic neuroendocrine tumours are a rare subgroup of pancreatic neoplasms. CASE REPORT We report on three patients having acute pancreatitis secondary to pancreatic neuroendocrine tumours, one of them with severe pancreatitis, and review the published cases up to now. Only 22 patients with acute pancreatitis sec...
متن کاملFamilial syndromes associated with neuroendocrine tumours
Neuroendocrine tumours may be associated with familial syndromes. At least eight inherited syndromes predisposing to endocrine neoplasia have been identified. Two of these are considered to be major factors predisposing to benign and malignant endocrine tumours, designated multiple endocrine neoplasia type 1 and type 2 (MEN1 and MEN2). Five other autosomal dominant diseases show more heterogene...
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ژورنال
عنوان ژورنال: Endocrine-Related Cancer
سال: 2010
ISSN: 1351-0088,1479-6821
DOI: 10.1677/erc-10-0024